Similarly, FLUAV load showed an elevated trend in in hiAT2 organoids co-infected with the omicron SARS-CoV-2 variant compared to hiAT2 organoids infected with FLUAV alone, although not statistically significant ( Figure 5 (A)).
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Reciprocal enhancement of SARS-CoV-2 and influenza virus replication in human pluripotent stem cell-derived lung organoids<sup>1</sup>.
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