Leaky Gut Plays a Critical Role in the Pathophysiology of Autism in Mice by Activating the Lipopolysaccharide-Mediated Toll-Like Receptor 4-Myeloid Differentiation Factor 88-Nuclear Factor Kappa B Signaling Pathway.

8 A–F), an overall trend of improvement in autistic behavior was consistently seen.

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Although there was no statistical difference in the expression of MyD88 and TLR4 between the two different treatments, DSS-treated BTBR mice showed an increasing trend of NF-κB compared to distilled water-treated mice (Fig. 9 D).

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