In addition, we explored the mechanism by which SchA exerts its anti-inflammatory effects on COPD model mice and found that SchA may modulate the Nrf2 signaling pathway and NLRP3 inflammasome activation, which may be significant in the treatment of COPD.
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Schisandrin A regulates the Nrf2 signaling pathway and inhibits NLRP3 inflammasome activation to interfere with pyroptosis in a mouse model of COPD.
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