Moreover, expression of CALB1 in cancer cells exhibited a highly significant inverse correlation with cancer cell–intrinsic expression of CXCL1 , CXCL2 , and CXCL8 and a positive correlation with CCL26 expression, which did not, however, reach statistical significance ( P = 0.057) ( Figure 6D ).
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Human endogenous retrovirus onco-exaptation counters cancer cell senescence through calbindin.
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