Eicosapentaenoic acid influences the pathogenesis of Candida albicans in Caenorhabditis elegans via inhibition of hyphal formation and stimulation of the host immune response.

This was consistent with results observed by Bouyanfif [ 60 ], where supplementation with EPA did not cause any significant change in EPA in wild-type nematodes, although a decreasing trend was seen for EPA as well as a non-significant increase in 18:3n-3, similar to our results.

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