Average unmyelinated axon diameter showed an increasing trend after injury in both models (Supplemental Figure 7D, available at http://links.lww.com/PAIN/B843 ), likely because of the presence of regenerating large fibres (see arrow heads in Supplemental Figure 7A, available at http://links.lww.com/PAIN/B843 ).
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Long-term tactile hypersensitivity after nerve crush injury in mice is characterized by the persistence of intact sensory axons.
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