Although the exact reason for the change from basic steatosis to NASH is unknown, the in vitro and in vivo studies project that chronic hyperglycemia-induced hepatocyte apoptosis is highly significant throughout this transition.
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Bioactivity-Guided Synthesis: In Silico and In Vitro Studies of <i>β</i>-Glucosidase Inhibitors to Cope with Hepatic Cytotoxicity.
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