Khk depletion restores structural synaptic plasticity by reducing microglial ROS production Our RNA-seq results showed that the abundance of synapse-related genes had a decreasing trend in the db/db mice compared with the m/m mice (Supplementary Fig. 14 ).
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Ketohexokinase-dependent metabolism of cerebral endogenous fructose in microglia drives diabetes-associated cognitive dysfunction.
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