The highly significant heritability of resistance in S. vulgaris that we demonstrated, combined with the severe fitness consequences of infection by the anther‐smut fungi, predicts a strong evolutionary response in the host phenotype under high disease pressure (Bruns et al., 2015 , 2019 ).
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Quantitative disease resistance in wild <i>Silene vulgaris</i> to its endemic pathogen <i>Microbotryum silenes-inflatae</i>.
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