We found that TM4SF5‐negative cells with or without FLAG‐FKBP8 overexpression did not show increased MLCS formation upon glucose repletion; however, HA‐TM4SF5‐positive cells showed insignificant ( P = 0.070) but an increasing trend in glucose‐mediated formation of MLCSs, which was further enhanced by FLAG‐FKBP8 overexpression (Figure 3H ).
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Glucose-mediated mitochondrial reprogramming by cholesterol export at TM4SF5-enriched mitochondria-lysosome contact sites.
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