The pancancer overexpressed NFYC Antisense 1 controls cell cycle mitotic progression through in cis and in trans modes of action.

7B ) and displayed a highly significant enrichment of TF binding sites (defined based on ChIP-seq signal and presence of consensus motif in the promoter) of complexes involved in G1/S gene repression ( i.e ., E2F4/5-DREAM) and G2/M gene activation ( i.e ., MYBL2, FOXM1) [ 44 ], with moderate or no enrichment for activator E2Fs or NF-Y, respectively (Fig. 7C and Supplementary Fig.

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