Investigating the role of an immediate early gene FOS as a potential regulator of autophagic response to hypoglycemia in embryonic hypothalamic neurons.

While FOS mRNA expression was hindered in the FOS KD cells across glucose conditions of 4500, 900, and 200 mg/L, BECN1 expression stayed low in the BECN1 KD cells at concentrations of 4500 and 900 mg/L but then showed an increasing trend at 200 mg/L, signifying the increased induction of autophagy through BECN1 expression at this concentration.

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