Cancer cells in high glucose conditions often exhibit an increasing trend in glucose uptake and glycolytic activity [ 49 , 50 ], and increased aerobic glycolysis intensifies the strength of immune suppression within tumors [ [51] , [52] , [53] , [54] ].
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Nuclear translocation of metabolic enzyme PKM2 participates in high glucose-promoted HCC metastasis by strengthening immunosuppressive environment.
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