The neuroimmune CGRP-RAMP1 axis tunes cutaneous adaptive immunity to the microbiota.

Of note, the most striking transcriptomic distinction between wild-type and Ramp1 -deficient commensal-induced Tc17 cells was observed in the most abundant Tc17 cluster (cluster 0), where the differentially expressed genes exhibited highly significant enrichment in factors controlling T cell activation ( Fig. 4 B ).

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