Targeting hepatic ceruloplasmin mitigates nonalcoholic steatohepatitis by modulating bile acid metabolism.

Consistently, Cp LKO mice showed significantly lower serum triglyceride (TG) ( Figure 2G ), total cholesterol (TC) ( Figure 2H ), and hepatic non-esterified fatty acid (NEFA) ( Figure 2J ) levels and a decreasing trend in serum NEFA level ( Figure 2I ) compared to Cp flox/flox mice.

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