Compared to H iHLCs, AC iHLCs exhibited a nonsignificant trend of 68% greater de novo lipogenesis (Fig. 2E ) and a significantly 47% greater fatty acid uptake ( P = 0.0029; Fig. 2F ).
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A patient-based iPSC-derived hepatocyte model of alcohol-associated cirrhosis reveals bioenergetic insights into disease pathogenesis.
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