We conducted a pan-cancer analysis on KIF26B and found that KIF26B was up-regulated in most cancers, with markedly significant differences in stomach adenocarcinoma (Figure 1C ). qRT - PCR demonstrated that KIF26B expression was higher in GC tissues than in normal tissues (Figure 1D ).
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Kinesin 26B modulates M2 polarization of macrophage by activating cancer-associated fibroblasts to aggravate gastric cancer occurrence and metastasis.
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