This hypothesized deficit should be larger, the longer is the delay between MOC loss and OHC death, leaving a more or less significant, transient population of orphaned OHCs that are no longer under efferent control.
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This hypothesized deficit should be larger, the longer is the delay between MOC loss and OHC death, leaving a more or less significant, transient population of orphaned OHCs that are no longer under efferent control.