21 , 42 Our data now indicate that both type I IFN activity and B-cell activity may be significant pathologic mechanisms in AQP4-NMOSD, but they may not play an equally significant role in MOGAD or SN-NMOSD patients.
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Serum Proteomics Distinguish Subtypes of NMO Spectrum Disorder and MOG Antibody-Associated Disease and Highlight Effects of B-Cell Depletion.
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