Increased ONECUT2 induced by Helicobacter pylori promotes gastric cancer cell stemness via an AKT-related pathway.

Further analyses of spatial transcriptomic data revealed an increasing trend in these four stemness markers in HP-infected GC tissue in the Po1 and Ne1 sections (Supplementary Fig. 5A–D ), and the Mki67 mRNA level was also elevated in HP-infected GC tissue (Supplementary Fig. 4G ).

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