Bioengineered Model of Human LGMD2B Skeletal Muscle Reveals Roles of Intracellular Calcium Overload in Contractile and Metabolic Dysfunction in Dysferlinopathy.

We, therefore, applied VAM and DNT to FA‐treated LGMD2B myobundles and found a slight trend for increased OCR (Figure S10A–E , Supporting Information), which, however, was accompanied by a significant increase in glycolysis and decrease in OCR:ECAR ratio (Figure S10F , Supporting Information).

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