As detailed in Table 1 , a highly significant enrichment in Alu S subfamily elements was observed among expressed Alu s in both control and dl 1500-infected cells, while Alu Y- and Alu J-derived transcripts were significantly underrepresented in Alu transcriptomes.
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Adenovirus small E1A directs activation of Alu transcription at YAP/TEAD- and AP-1-bound enhancers through interactions with the EP400 chromatin remodeler.
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