Proteomics identifies potential immunological drivers of postinfection brain atrophy and cognitive decline.

Despite limited power, sensitivity analyses restricting the comparison group to participants with a history of no infections (for example, influenza versus no history of any infection) showed consistent atrophy linked to influenza, herpes viruses, miscellaneous viral infections and LRTIs, with URTI- and skin and subcutaneous infection-related atrophy remaining marginally significant ( P ≤ 0.10) ( Supplementary Table 4 ).

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