As a main finding ( Figure 2 a,b), there was a highly significant more than two-fold PDAC-related increase in percentage of atrogin-1+ cells (atrogin-1+ cells in % of hematoxylin+ cells) in myocardial cross-sections.
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Increased Myocardial MAO-A, Atrogin-1, and IL-1β Expression in Transgenic Mice with Pancreatic Carcinoma-Benefit of MAO-A Inhibition for Cardiac Cachexia.
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