indicated that there were highly significant correlations among the glomerular filtration rate, inflammation, and lipid metabolism genes, evidencing the possible role of abnormal lipid metabolism in the pathogenesis of DM [ 46 ].
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Molecular mechanism of ectopic lipid accumulation induced by methylglyoxal via activation of the NRF2/PI3K/AKT pathway implicates renal lipotoxicity caused by diabetes mellitus.
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