Furthermore, density maps, depicting the cell distribution of dMMR and pMMR with the pseudotime, demonstrated that towards the end of the differentiation process, there was an increasing trend in pMMR patients for GC2 cells, whereas a decreasing trend was observed in dMMR patients (Fig. 2 L).
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SPP1+ macrophages and FAP+ fibroblasts promote the progression of pMMR gastric cancer.
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