KCASP1Tg mice, the mouse model used in this study, combine the pathogenesis of AD and psoriasis, and it is highly significant to examine the effect of IL-17 deficiency on the suppression of complications in this model.
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Amelioration of Systemic Amyloidosis by Blocking IL-17A and Not by IL-17F, and Arteriosclerosis by Blocking Both IL-17A and IL-17F in an Inflammatory Skin Mouse Model.
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