Interruption of glucagon signaling augments islet non-alpha cell proliferation in SLC7A2- and mTOR-dependent manners.

[ 26 ] showed that treatment of C57BL6 mice with a GCGR-Ab at a weekly dose of 5 mg/kg for 4 weeks increased islet delta cell number by ∼35% in association with marginally significant (P = 0.05) delta cell proliferation.

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