Inaccurate estimations of Na intake due to random/systematic errors [ 11 ], inconsistent definitions [ 12 , 13 ], and a failure to consider diverse phenotypes of HTN [ 14 , 15 ] in population risk assessment may be significant sources of controversial evidence, including artefactual J‐shaped, U-shaped, and inverse relationships between salt intake and risk of HTN [ 16 , 17 , 18 ].
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The association between urine-estimated salt intake and hypertension: findings of a population-based study.
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