Pharmacological Inhibition of N-Acylethanolamine Acid Amidase (NAAA) Mitigates Intestinal Fibrosis Through Modulation of Macrophage Activity.

NAAA upregulation was associated with a significant decrease in PEA ( Figure 1I ) and OEA levels ( Figure 1J ) (ie, its preferential substrates), with a nonsignificant trend toward the decrease in AEA levels ( Figure 1K ).

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