Axonal transport of CHMP2b is regulated by kinesin-binding protein and disrupted by CHMP2b<sup>intron5</sup>.

Although BIC/4AP led to a highly significant increase in the density of PLA puncta for WT mCh-CHMP2b– versus mCh-CHMP2b intron5 –expressing neurons ( Fig 7C–F ), this treatment did not significantly increase the number of puncta in WT mCh-CHMP2b–expressing neurons compared with the control condition (although the data trend in this direction; Fig 7F ).

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