Different types of cell death and their interactions in myocardial ischemia-reperfusion injury.

Another experiment also clearly showed that Drp1 has an increasing trend in myocardial I/R injury, and vitexin can reduce myocardial damage by affecting mitochondrial function through the reduction of Drp1 expression [ 66 ].

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However, there may be significant differences in the cell death regulatory mechanisms among different patients with myocardial I/R injury, which makes the precise treatment plan based on the synergy of cell death pathways highly individualized and increases the complexity of treatment.

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The correlation between mitochondrial function and necroptosis is highly significant.

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