Accurate estimation of the burden of LF is difficult due to the considerable variability in the incidence of chronic liver diseases in different geographical regions and the paucity of high-quality epidemiological data; however, the global burden of LF-involved chronic liver diseases is unequivocally substantial and shows an increasing trend due to the rising prevalence of unhealthy lifestyles [ 1 ].
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Nedd4L signaling contributes to carbon tetrachloride-induced liver fibrosis in female mice and is associated with enteric dysbacteriosis.
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