Mechanisms of rifaximin inhibition of hepatic fibrosis in mice with metabolic dysfunction associated steatohepatitis through the TLR4/NFκB pathway.

Similarly, western blot showed a significant increase in α-SMA content in the FFA group; the low-dose intervention group (RFX-D) showed a decreasing trend in α-SMA protein expression but no statistical difference; in the medium- and high-dose groups (RFX-Z, RFX-H), the decrease was obvious after rifaximin intervention (Fig. 7 G).

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