In CAA, there was a highly significant doubling in a minor HS component, unsubstituted glucosamine (0.7% vs 1.4% by mole percentage), which is normally present in the vasculature. 30 This increase was largely driven by D0H0.
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Multi-omic analysis of meningeal cerebral amyloid angiopathy reveals enrichment of unsubstituted glucosamine and extracellular proteins.
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