Further, a transcriptome-wide association study (TWAS) from our group identified KLHL17 as a borderline significant gene in the pancreas and multi-tissue models (Z = −3.96, FDR = 0.052; FDR = 0.063, respectively) 28 .
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Allelic effects on KLHL17 expression underlie a pancreatic cancer genome-wide association signal at chr1p36.33.
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