In addition, retest 2 showed an almost significantly faster escape latency for TG mice with GAL-201 treatment (AUC = 0.3 [0.08; 0.55]).
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GAL-201 as a Promising Amyloid-β-Targeting Small-Molecule Approach for Alzheimer's Disease Treatment: Consistent Effects on Synaptic Plasticity, Behavior and Neuroinflammation.
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