The role of SPI1/VSIG4/THBS1 on glioblastoma progression through modulation of the PI3K/AKT pathway.

J Adv Res · 2025 · PMC12126732 · PMID 38960279

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a decreasing trendno p-value reported

Following the knockdown of VSIG4, the total levels of STAT3 in both LN229 and U251 cell lines remained unchanged, while the phosphorylated form of STAT3 exhibited a decreasing trend.

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