Notably, Acer3 upregulation was observed at 6 hours of reperfusion in I/R livers and showed a decreasing trend by 24 hours, although it remained higher than sham levels ( Figure 1, K–M ).
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Ceramide(d18:1/18:1)-NDUFA6 interaction inactivates respiratory complex I to attenuate oxidative-stress-driven pathogenesis in liver ischemia/reperfusion injury.
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