Speckle fluctuations reveal dynamics of microparticles in fibrin scaffolds in a model of bacterial infection.

Whereas these two mechanisms discussed above (polymerization-incurred stress and syneresis) are governed by the fibrin network itself, an increasing trend in α III is observed as a function of increasing bead size (Fig. 2j , P = 6.5 × 10 –4 ), indicating that the particles also participate in driving the extent of the observed super-diffusive dynamics.

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As the mesh size decreases over the course of polymerization, the increasing steric hindrance results in progressively more sub-diffusive dynamics, corresponding to a decreasing trend in α I as a function of Δ t .

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