Altered protein homeostasis in cardiovascular diseases contributes to Alzheimer's-like neuropathology.

Terms with enrichment scores > 4 are considered highly significant [ 113 ]; these included ATP-binding, microtubule binding, pleckstrin homology (conferring the potential to bind PIP 3 ), ubiquitin-like protein conjugation, cytoskeleton, RNA binding, kinases, pathways of neurodegeneration, etc., each with Benjamini P < 1.7 × 10 ‒ 5 .

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