Loss of LAPTM4A inhibits M2 polarization of tumor-associated macrophages in glioblastoma, promoting immune activation and enhancing anti-PD1 therapy.

However, in recent years, there has been an increasing trend in research focusing on immune cells within the glioma microenvironment 64 , 65 .

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In this context, gaining a comprehensive understanding of key regulatory factors is highly significant, particularly about the function and regulation of TAMs in the tumor microenvironment to advance novel treatment strategies for Glioma.

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