Barely Significant
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Tempol Induces Oxidative Stress, ER Stress and Apoptosis via MAPK/Akt/mTOR Pathway Suppression in HT29 (Colon) and CRL-1739 (Gastric) Cancer Cell Lines.

Curr Issues Mol Biol · 2025 · PMC12293240 · PMID 40729043

Europe PMCDOI
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hedged sentence
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closest p · 0.0× alpha
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The sentences

highly significantp < 0.0001actually significant
Tempol Promotes Apoptosis of HT29 and CRL1739 Cells by Modulating Pro-Apoptotic and Anti-Apoptotic Proteins As presented in Table 1 , Tempol treatment resulted in a highly significant ( p < 0.0001) increase in Bax and cleaved caspase-3 protein levels compared to the control groups in both gastric (CRL1739) and colon (HT29) cancer cells.

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