NR4A1 Mediates Bronchopulmonary Dysplasia-Like Lung Injury Induced by Intrauterine Inflammation in Mouse Offspring.

Similarly, treatment with EREG also resulted in a decreasing trend in E-CADHERIN expression, which could be effectively rescued by gefitinib ( Figure 5 k), suggesting that NR4A1 may regulate EMT through a mechanism partially dependent on the EREG–EGFR signaling pathway. 3.

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