Interestingly, we found that after stable overexpression of NXF1, extremely significant variable alternative splicing events occurred in cells, and a large proportion of these alternative splicing events were found to be Exon skipping after analysis ( Figures 3 A and 3B).
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NXF1 suppresses progression of endometrial cancer by interacting with the SRSF3 to regulate SP4 splicing.
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