Compared with NC, eigengene expression was significantly elevated in AD but showed a decreasing trend in CAA ( Fig. 3C ), suggesting that astrocytes may promote AD progression by damaging neuronal synapses and contribute to CAA through effects on endothelial cell functions.
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Differential Roles of Astrocytic CSF1 in Alzheimer's Disease and Cerebral Amyloid Angiopathy: Insights from Transcriptomic Analysis.
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