Baicalein ameliorates DSS-induced ulcerative colitis in mice by inhibiting ferroptosis and regulating gut microbiota.

Although ACSL4 expression exhibited a decreasing trend in our current study, Nrf2 and SLC7A11 showed an increasing trend, particularly pronounced in the high-dose baicalein cohorts.

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Particularly noteworthy, KEGG pathway analysis (S5) showed highly significant enrichment of the ferroptosis pathway.

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