DPP4-inhibition reduces pro-inflammatory cytokine production by alpha-beta and gamma-delta T cells in vitro and in the biliary atresia mouse model.

In those, DPP4 inhibition also led to a dose-dependent, highly significant decreased production of IFN-γ and IL-17 with an about 12-fold reduction of IFN-γ and a 7-fold reduction in IL-17 concentration (Fig. 3 D).

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