Since CLR susceptibility is a strong predictor of treatment outcome among MAB infections, an increasing trend of CLR resistance, including inducible resistance, is alarming, and early identification is empirical for treating MAB infections ( 36 , 37 ).
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Luciferase reporter mycobacteriophage (TM4::<i>GeNL</i>) enables rapid assessment of drug susceptibilities and inducible macrolide resistance in <i>Mycobacterium abscessus</i> complex.
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