Naringenin Alleviates the Autophagy-Associated AMPK-Akt/mTOR Pathway to Regulate Inflammation and Barrier Function, Decreasing DSS-Induced Intestinal Fibrosis.

Exposure to DSS resulted in highly significant increases (2.59-fold and 1.07-fold, respectively; p < 0.05) in the serum concentrations of D-LA and LPS in colitis mice, indicating that intestinal mucosal barrier function was disrupted.

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